My lab is interested in how the innate immune response prioritizes what it responds to when faced with multiple simultaneous insults. This includes understanding how influenza A virus impacts the response to a secondary pulmonary infection with bacteria, and how pulmonary infections impact the ability to heal a cutaneous wound. Using several complementary models we have shown that lung infections cause a rapid, sustained, chemokine-dependent suppression of the innate immune wound healing response, and subsequently a delay in cutaneous wound healing. These data demonstrate a new biological premise of regulated immune prioritization.